Skip to main content

why you see what I see...for the most part....

I find that running is an excellent activity to be engaged in when I need my mind to wander, the mind walks I've taken while so engaged have revealed the solution to many difficult problems in my code and design of a distributed web application framework. The freedom of thought inspired by this part of my day often inspires entirely unrelated ideas to my current line of work. Case in point was an idea that I explored while running several months ago. As I concentrated on breathing and keeping my form optimal for my pace, I took a moment to fully experience the vivid colors of the city and of nature that passed me as I ran. I thought to myself of an idea that I'd come across much earlier in my life as a high school student but then had not the knowledge to answer the question.

The question is thus: Why do you and I experience color the same way?

Having gained knowledge on how the human visual system works, as well as how the brain-eye system processes visual stimulation the answer follows from simply traversing the path of photons as they enter the eye. The journey proceeds as follows. The first stage involves photons of various energies released or reflected by the surrounding environment in the direction of the eye. Electromagnetic theory tells us that photon energy corresponds to photon wavelength in a definite way by way of the equation:

E = hv

where E is the energy , h is the Planck constant and v is not the Roman "vee" but rather the greek symbol "nu" and represents the frequency of the propagating photon. Since frequency is inversely related to wavelength , the equation could be written as:

E= h/w

where w is now the wavelength of the propagating photon. I won't get into the potentially sticky area of photons as propagating particles versus propagating waves, Physicists learned that in order for electromagnetic equations to provide consistent results both interpretations are required depending on what is happening at the interaction point. For our journey the interaction occurs at the back of the human retina, but before we get there, a bit about the wavelength and energy. It just so happens that photons with high frequency (therefore low wavelength) have higher energy than photons with low frequency or high wavelength. In my time studying quantum mechanics I've conceptualized this as a measure of the interaction probability for the propagating photon. High energy photons have high interaction probabilities while lower ones have lower interaction probabilities...unfortunately this fails to hold. The curious nature in which matter absorbs energy in quantized packets means that it is possible for high frequency photons to be completely ignored by certain materials if the quantum states required to stimulate those materials are not triggered. In particular, the mechanism of stimulation employed by eyes involves stimulation of photosensitive enzymes in the rod and cone sensors in the back of our retinas. The rods are sensitive to ANY photon energy while the cones have specific filter ranges of sensitivity. Like the aformentioned surfaces, the enzymes that form the sensitive material of these rods and codes reacts in specific ways. The rods are sensitive to a plethora of energies across the electromagnetic spectrum thus making them good gauges of intensity, where the cones have specific violet, red-blue and yellow-green sensitivity ranges. The perception of a rainbow of colors comes from our brains synthesis of the many signals coming from these tiny cones. These sensitivites are provided by enzymes that have specific molecular construction that is stimulated when photons of given energies or ranges impinge them. The molecules absorb part of the energy and emit additional photons that propagate down the optic nerve in a cascade of triggered stimulation and emmissions. Eventually these signals reach the brains visual system and are synethesized into the perceptions of color that we have.

The answer to the question of why we see the same is in fact hidden in the mechanism of transition. The cones consist of specific types, that will be stimulated only when photons having the required energy impacts them, the mechanism for this is explained by quantum jumps of photons from high to low orbital positions. These gap jumps are always the same because the unique molecules are constructed of the exact same elements. Thus a 445 nm photon hitting a cone sensitive to that wavelength of energy will always kick an electron to a different orbital and in so doing release a photon of a specific energy in return...thus continuing the cascade. The point is the underlying physics of the molecules is what normalizes the response to the original photons at the rod and cone sensitive surfaces. All photons that do not induce stimulation at a cone are filtered out of the response leaving only the pure responses to fixed energies. Since these responses are tied to the enzymes used to transmit the signal to the brain, and these enzymes are the same ones used in all human beings , the response at the brain processing region must be the same. However this is not sufficient to conclude that the "perception" of stimuli will be the same. Here is the consistency of neuronal cells comes into play, whatever neurotransmitters that are responsible for relaying the visual signals (now normalized) they cause the same effects at the neurons, so long as the quantities of neuronal stimulation are similar the perception will be the same. Note the key phrase "so long as", there are in fact examples of perception being different for different people. People with color blindness for example see a skewed pallette as a result of a different filtering of the stimuli at the rods and cones, similarly variations in the neurotransmitters between people can similarly lead to warped perceptions (as may be experienced by those under the effects of psychoactive drugs or other brain modifying agents) so the answer is not as cut and dried as it may seem to be from the outset. Perception does change when ever any stage of the stimulation, neurotransmission or neuronal processing is effected, otherwise the preeminence of the physics (the underlying photon stimulated emission cascades) leads to similar perceptions for all.




http://hyperphysics.phy-astr.gsu.edu/hbase/vision/rodcone.html#c2

http://hubel.med.harvard.edu/b40.htm

Comments

Popular posts from this blog

the attributes of web 3.0...

As the US economy continues to suffer the doldrums of stagnant investment in many industries, belt tightening budgets in many of the largest cities and continuous rounds of lay offs at some of the oldest of corporations, it is little comfort to those suffering through economic problems that what is happening now, has happened before. True, the severity of the downturn might have been different but the common factors of people and businesses being forced to do more with less is the theme of the times. Like environmental shocks to an ecosystem, stresses to the economic system lead to people hunkering down to last the storm, but it is instructive to realize that during the storm, all that idle time in the shelter affords people the ability to solve previous or existing problems. Likewise, economic downturns enable enterprising individuals and corporations the ability to make bold decisions with regard to marketing , sales or product focus that can lead to incredible gains as the economic

How many cofactors for inducing expression of every cell type?

Another revolution in iPSC technology announced: "Also known as iPS cells, these cells can become virtually any cell type in the human body -- just like embryonic stem cells. Then last year, Gladstone Senior Investigator Sheng Ding, PhD, announced that he had used a combination of small molecules and genetic factors to transform skin cells directly into neural stem cells. Today, Dr. Huang takes a new tack by using one genetic factor -- Sox2 -- to directly reprogram one cell type into another without reverting to the pluripotent state." -- So the method invented by Yamanaka is now refined to rely only 1 cofactor and b) directly generate the target cell type from the source cell type (skin to neuron) without the stem like intermediate stage.  It also mentions that oncogenic triggering was eliminated in their testing. Now comparative methods can be used to discover other types...the question is..is Sox2 critical for all types? It may be that skin to neuron relies on Sox2

AgilEntity Architecture: Action Oriented Workflow

Permissions, fine grained versus management headache The usual method for determining which users can perform a given function on a given object in a managed system, employs providing those Users with specific access rights via the use of permissions. Often these permissions are also able to be granted to collections called Groups, to which Users are added. The combination of Permissions and Groups provides the ability to provide as atomic a dissemination of rights across the User space as possible. However, this granularity comes at the price of reduced efficiency for managing the created permissions and more importantly the Groups that collect Users designated to perform sets of actions. Essentially the Groups serve as access control lists in many systems, which for the variable and often changing environment of business applications means a need to constantly update the ACL’s (groups) in order to add or remove individuals based on their ability to perform cert